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Phosphorylation of spore coat proteins by a family of atypical protein kinases

Table S2.

B. subtilis strains used in this study

Strain Genotype or description Reference or construction*
PY79 Wild-type laboratory strain (73)
ΔcotH ΔcotH::kanr pcotH::kanr → PY79 (KmR)
CotH-V5 (PcotH) ΔcotH::kanr amyE::PcotG-PcotH-cotH-V5 pJLG13-PcotG-PcotH-cotH-V5 → ΔcotH (KmR) (CmR)
CotHD251A-V5 (PcotH) ΔcotH::kanr amyE::PcotG-PcotHD251A-cotH-V5 pJLG13-PcotG-PcotH-cotHD251A-V5 → ΔcotH (KmR) (CmR)
CotHD251A-V5 (PcotA) ΔcotH::kanr amyE::PcotA-PcotHD251A-cotH-V5 pamyE::PcotA-cotHD251A-V5 → ΔcotH (KmR) (CmR)
CotHD251A-V5 (PcotE) ΔcotH::kanr amyE::PcotE-PcotH-cotHD251A-V5 pamyE::PcotE-cotHD251A-V5 → ΔcotH (KmR) (CmR)
CotHD251A-V5 (PgerE) ΔcotH::kanr amyE::PgerE-PcotH-cotHD251A-V5 pamyE::PgerE-cotHD251A-V5 → ΔcotH (KmR) (CmR)
  • * Plasmid or genomic DNA used (right of the arrow) to transform an existing strain (left of the arrow) to create a new strain is listed. The drug resistance is noted in parentheses. CmR, chloramphenicol resistance; KmR, kanamycin resistance.

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